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A cell-permeable chemical inhibitor of p53.
DMSO: 100 mg/mL
Murphy, P.J.M., et al. 2004. J. Biol. Chem.279, 30195. Culmsee, C., et al. 2001. J. Neurochem.77, 220. Komarova, E.A., and Gudkov, A.V. 2000. Biochemistry (Mosc.) 65, 41. Stadler, P., et al. 2000. Strahlenther Onkol. 176, 98. Ferber, D. 1999. Science 285, 1651. Jacquemin-Sablon, A. 1999. Bull. Cancer86, 894. Komarov, P.G., et al. 1999. Science 285, 1733.
A reversible inhibitor of p53-dependent transactivation of p53-responsive genes and reversibly blocks p53-mediated apoptosis. Inhibits p53-dependent growth arrest of human diploid fibroblasts in response to DNA damage but has no effect on p53-deficient fibroblasts. Protects mice from lethal genotoxic stress associated with anticancer treatment without promoting tumor formation. Has been reported to protect neurons against β-amyloid and glutamate-induced apoptosis.
Packaged under inert gas
Reversible: yes
Following reconstitution, aliquot into cold tubes and freeze (-20°C). Stock solutions in DMSO are stable for 3 months at -20°C. Unstable in aqueous solutions.
Toxicity: Standard Handling (A)
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